ECG in PoisoningECG in poisoning refers to changes in waves, intervals, and rhythm caused by the toxic effects of drugs or poisons on the heart's ion channels and conduction system.
Digoxin Effect on ECGThe digoxin effect refers to characteristic changes in the ST segment, T wave, and QT interval associated with the use of cardiac glycosides.
Digoxin ToxicityDigoxin toxicity (digitalis toxicity) is the toxic effect of cardiac glycosides on the myocardium and conduction system resulting from drug overdose or accumulation.
Tricyclic Antidepressant PoisoningTricyclic antidepressant (TCA) poisoning is the cardiotoxic effect of TCA overdose due to the blockade of myocardial fast sodium channels.
Sodium Channel Blocker PoisoningSodium channel blocker poisoning is the toxic inhibition of fast sodium channels in cardiomyocytes by drugs or poisons, which slows depolarization.
Beta-Blocker PoisoningBeta-blocker poisoning (beta-blocker toxicity) is acute intoxication with drugs that block β-adrenoreceptors, resulting in the suppression of automaticity and conduction.
Calcium Channel Blocker PoisoningCalcium channel blocker poisoning (CCB toxicity) is an overdose of verapamil, diltiazem, or dihydropyridines (such as amlodipine) leading to the depression of…
Carbamazepine CardiotoxicityCarbamazepine cardiotoxicity refers to cardiac conduction and rhythm disorders during carbamazepine overdose due to the blockade of fast sodium channels.
Quetiapine PoisoningQuetiapine poisoning (quetiapine toxicity) is an overdose of an atypical antipsychotic affecting potassium channels and, to a lesser extent, sodium channels in cardiomyocytes.
QT-Prolonging DrugsQT-prolonging drugs are medications that delay ventricular repolarization and prolong the QT interval on the ECG.
Antiarrhythmics on ECGAntiarrhythmics on ECG refers to characteristic interval and wave changes caused by antiarrhythmic drugs; the effects of Vaughan-Williams classes IA, IC, and III are primarily considered below…
Cocaine and ECGCocaine and ECG refers to the combination of the toxic effects of cocaine on the myocardium and conduction system, which can mimic or trigger a true acute coronary syndrome.
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