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Neurology

Chapter 15. Meningeal and Hypertensive Syndromes, Hydrocephalus

3 topics with a brief review: definitions, key facts, and class questions.

This chapter is dedicated to critical conditions in neurology caused by meningeal irritation and impaired CSF circulation. The ability to quickly recognize these pathologies is a core skill for a physician of any specialty, as time often runs out in hours and minutes.

The section consists of three key topics: meningeal syndrome, hypertensive syndrome, and hydrocephalus. We will analyze the pathogenesis of meningeal signs, mechanisms of intracranial pressure elevation, and modern classifications of hydrocephalus.

During practical classes and exams, you will certainly be asked to demonstrate classic meningeal signs (nuchal rigidity, Kernig's and Brudzinski's signs). Most frequently, instructors ask about the differential diagnosis of communicating and non-communicating (occlusive) hydrocephalus, the differences between meningism and true meningitis, as well as characteristic fundoscopic changes (papilledema) in intracranial hypertension.

Section Topics

Frequently asked questions for this section

Where exactly does cerebrospinal fluid circulate?

CSF circulates in the cerebral ventricles and in the subarachnoid space, which is located between the arachnoid mater and the pia mater.

Why does intracranial hypertension occur in adults?

In adults, the skull bones are firmly fused and non-distensible. Any increase in the volume of intracranial contents inevitably raises the pressure.

Why do the ventricles not dilate significantly in aresorptive hydrocephalus?

When CSF resorption is impaired, CSF pressure rises on the outside of the brain. This "external" hypertension mechanically prevents excessive ventricular dilation from within.

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