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Cardiology

ECG Interpretation: Norm, Waves, Intervals, Algorithm

WhiteCoat Pro Editorial BoardApril 11, 2026Updated September 16, 20266 min read
Contents · 12 sections
  1. 1. Technical Fundamentals
  2. 2. Leads and Their Axes
  3. 3. Waves and Normal Values
  4. 4. Electrical Axis of the Heart
  5. 5. Heart Rate Calculation
  6. 6. Sinus Rhythm
  7. 7. Conduction Blocks
  8. 8. Hypertrophy
  9. 9. Ischemia and Infarction
  10. 10. Interpretation Algorithm
  11. 11. Electrolyte Disturbances on ECG
  12. 12. FAQ

In a nutshell. ECG is a graphical recording of the electrical activity of the heart. Standard 12-lead ECG: 6 chest leads (V1–V6) + 3 standard limb leads (I, II, III) + 3 augmented limb leads (aVR, aVL, aVF). Paper speed 25 mm/s (1 mm = 0.04 s), 10 mm = 1 mV. Norm: P < 0.12 s and < 2.5 mm, PR 0.12–0.20 s, QRS 0.06–0.10 s, QT (corrected) ≤ 0.44 s in men and ≤ 0.46 s in women, HR 60–100 bpm, axis −30° to +90°. Algorithm: rhythm → HR → axis → intervals → waves → ST segment → ischemic/infarct changes → conduction disturbances.

1. Technical Fundamentals

  • Recording speed 25 mm/s (50 mm/s can be used for greater detail).
  • Calibration: 10 mm = 1 mV (standard).
  • On standard grid paper: 1 small square = 1 mm = 0.04 s; 1 large square = 5 mm = 0.20 s.
  • Lead placement: limb electrodes (red — right arm, yellow — left arm, green — left leg, black — right leg / ground).
  • Chest leads V1–V6: V1 — 4th intercostal space at the right sternal border, V2 — 4th intercostal space at the left sternal border, V4 — 5th intercostal space at the midclavicular line, V3 — midway between V2 and V4, V5 — anterior axillary line at the level of V4, V6 — midaxillary line at the level of V4.

2. Leads and Their Axes

LeadAxis (°)Myocardial Region
I0°Lateral wall of LV
II+60°Inferior wall
III+120°Inferior wall
aVR−150°LV cavity (reciprocal)
aVL−30°Anterolateral
aVF+90°Inferior wall
V1–V2—Interventricular septum, right ventricle
V3–V4—Anterior wall
V5–V6—Lateral wall

3. Waves and Normal Values

  • P wave — atrial depolarization; positive in I, II, aVF, V2–V6; amplitude < 2.5 mm; duration < 0.12 s.
  • PR interval — from the onset of P to the onset of QRS; 0.12–0.20 s.
  • QRS complex — ventricular depolarization; 0.06–0.10 s.
  • R wave — main positive wave; progressive increase in amplitude from V1 to V5–V6.
  • S wave — deep in V1–V2, diminishing towards V5–V6.
  • Q wave — < 0.03 s and < 1/4 of R wave amplitude (in leads other than aVR, V1); pathological if > 0.04 s or deeper.
  • ST segment — isoelectric; elevation/depression > 1 mm is considered abnormal.
  • T wave — ventricular repolarization; positive in most leads except aVR and V1 (variable).
  • QT interval — from the onset of QRS to the end of T; Bazett's correction formula QTc = QT / √RR.
  • U wave — small positive deflection following the T wave; not always visible, associated with hypokalemia.

4. Electrical Axis of the Heart

  • Norm: from −30° to +90° (most commonly 0° to +60°).
  • Left axis deviation (LVH / LBBB): from −30° to −90°.
  • Right axis deviation (RVH / PE / left posterior fascicular block): from +90° to +180°.
  • Extreme axis deviation (−90° to ±180°): "northwest quadrant" — rare, indicative of pathology.
  • Rapid method: QRS in I and aVF. Both positive — normal; I negative, aVF positive — right axis deviation; I positive, aVF negative — left axis deviation.

5. Heart Rate Calculation

  • Regular rhythm: 1500 / (number of small squares between R-R) or 300 / (number of large squares).
  • Large squares between R-R: 1 → 300, 2 → 150, 3 → 100, 4 → 75, 5 → 60, 6 → 50.
  • Irregular rhythm: count the number of QRS complexes in 6 seconds (30 large squares) and multiply by 10.
  • Norm 60–100 bpm; bradycardia < 60, tachycardia > 100.

6. Sinus Rhythm

  • Regular P waves preceding each QRS complex, identical in morphology.
  • P wave positive in I, II, aVF, negative in aVR.
  • PR interval constant and within normal limits.
  • HR 60–100 bpm (< 60 — sinus bradycardia; > 100 — sinus tachycardia).
  • Sinus (respiratory) arrhythmia in young individuals and athletes — normal variant.

7. Conduction Blocks

  • First-degree AV block: PR > 0.20 s.
  • Second-degree AV block, Mobitz type I (Wenckebach): progressive PR prolongation until a QRS complex is dropped.
  • Mobitz type II: constant PR interval with intermittent dropped QRS complexes without prior prolongation.
  • Third-degree AV block (complete heart block): atria and ventricles beat independently.
  • RBBB (Right Bundle Branch Block): rSR' in V1–V2, wide S in I, V5–V6; QRS > 0.12 s.
  • LBBB (Left Bundle Branch Block): wide mono- or biphasic R in I, aVL, V5–V6; QS or rS in V1–V2; QRS > 0.12 s.
  • Incomplete RBBB — QRS 0.10–0.12 s with rSR' in V1.

8. Hypertrophy

  • Left ventricular hypertrophy (Sokolow-Lyon): S(V1) + R(V5 or V6) ≥ 35 mm.
  • Cornell index: R(aVL) + S(V3) > 28 mm in men, > 20 in women.
  • Romhilt-Estes — point score system.
  • Right ventricular hypertrophy: R/S > 1 in V1, deep S in V5–V6, right axis deviation.
  • Right atrial hypertrophy: "P-pulmonale" — tall peaked P ≥ 2.5 mm.
  • Left atrial hypertrophy: "P-mitrale" — notched wide P in I, II; negative phase in V1 deeper than 1 mm and wider than 0.04 s.

9. Ischemia and Infarction

  • Subendocardial ischemia — ST depression ≥ 1 mm, horizontal/downsloping.
  • Subepicardial ischemia — ST elevation ≥ 1 mm in two contiguous leads (in V2–V3 — ≥ 2 mm in men, 1.5 mm in women).
  • MI zones by leads: inferior (II, III, aVF → RCA), anterior (V1–V4 → LAD), lateral (I, aVL, V5–V6 → LCx), posterior (depression in V1–V3, reciprocal picture; V7–V9 — elevation), RV (V3R, V4R).
  • MI dynamics: ↑ ST (minutes-hours) → pathological Q (hours-days) → inverted T (weeks-months) → normalization.
  • Reciprocal depression — confirms STEMI.

10. Interpretation algorithm

  • 1. Sinus rhythm? (regular P before each QRS, positive in II).
  • 2. Heart rate (60–100?).
  • 3. Electrical axis.
  • 4. Intervals: PR, QRS, QT/QTc.
  • 5. P wave (amplitude, duration, morphology).
  • 6. QRS complex (amplitude, morphology, presence of pathological Q waves).
  • 7. ST segment (elevation, depression).
  • 8. T wave (amplitude, inversion, symmetry).
  • 9. Conduction abnormalities.
  • 10. Comparison with previous ECGs.

11. Electrolyte disorders on ECG

  • Hyperkalemia: tall peaked T waves → PR prolongation → QRS widening → sine wave pattern → asystole.
  • Hypokalemia: flat T wave, prominent U wave, ST depression, QT prolongation.
  • Hypercalcemia: shortened QT.
  • Hypocalcemia: QT prolongation, without T wave changes.
  • Hypothermia: Osborn wave (J wave) — positive at the J point, most commonly in V3–V6.

12. FAQ

Why does aVR show ventricular processes in a mirror image?
The axis of aVR is directed from the heart to the right shoulder (−150°), i.e., opposite to the main LV depolarization vector. Therefore, all main waves are inverted: P is negative, R is small, Q is deep, T is normally negative.

What is QTc and why is it corrected?
QT duration depends on heart rate — the faster the rhythm, the shorter the QT. For standard comparison, QTc is used (Bazett: QT/√RR; Fridericia for rapid rhythms). Normal is < 0.44 s (M), < 0.46 (F); > 0.50 indicates a high risk of Torsades de Pointes.

When are additional leads V7–V9 and V3R–V4R needed?
V7–V9 — when posterior MI is suspected (with ST depression in V1–V3 and high R). V3R–V4R — during inferior MI to rule out right ventricular involvement (ST elevation in V4R is a marker).

How to quickly distinguish sinus tachycardia from AFib?
Sinus tachycardia is regular, with a P wave before each QRS. Atrial fibrillation is irregular, lacking P waves, with chaotic "f" waves and varying R-R intervals.

Почему важна сравнительная ЭКГ?
Many changes can only be interpreted in dynamics. New ST elevation or the appearance of Q waves indicates an MI; chronic changes indicate prior MI. An isolated "reciprocal" picture without old ECGs may be mistaken for normal.

Смотрите также: Myocardial infarction · Arrhythmias · ECG blocks · Hyperkalemia · Cardiology catalog

This material is educational in nature and does not replace a doctor's consultation. Verify treatment regimens and dosages with current clinical guidelines.

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